Skip to main content
Fig. 6 | Journal of Neuroinflammation

Fig. 6

From: Inhibition of neutrophil extracellular traps alleviates blood–brain barrier disruption and cognitive dysfunction via Wnt3/β-catenin/TCF4 signaling in sepsis-associated encephalopathy

Fig. 6

The expression distribution of the top 1 modules associated proteins in the hippocampus. A Double labeling IF staining of VCL with microglia (Iba-1), astrocytes (GFAP), neurons (NeuN) and ECs (CD31) in the hippocampus (n = 6). B The colocalization analysis of VCL with Iba-1, GFAP, NeuN and CD31 by ImageJ (n = 6). C Double labeling IF staining of Fermt1 with microglia (Iba-1), astrocytes (GFAP), neurons (NeuN) and ECs (CD31) in the hippocampus (n = 6). D The colocalization analysis of Fermt1 with Iba-1, GFAP, NeuN and CD31 by ImageJ (n = 6). NETs: Neutrophil extracellular traps; CLP: Cecal Ligation and Puncture; SAE: Sepsis-associated encephalopathy; IF: immunofluorescence

Back to article page

Journal of Neuroinflammation

ISSN: 1742-2094