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Fig. 1 | Journal of Neuroinflammation

Fig. 1

From: New insights into metabolism dysregulation after TBI

Fig. 1

Cellular metabolism dysregulation in TBI. TBI-induced changes of canonical glucose metabolism including increased reliance on lactate and glycolytic metabolism, lipid derived metabolites, increased oxidative stress, inflammation and altered mitochondrial function are schematically displayed. ATP: Adenosine triphosphate; BBB: Blood Brain Barrier leakage; BCAA: Branched Chain Amino Acid; GLUTs: Glucose transporters; Cyt C: Cytochrome C; FAD: Flavin adenine dinucleotide; NAD: Nicotinamide adenine dinucleotide; Nrf2: Nuclear factor erythroid 2-related factor 2; PLA2: Phospholipase A2; ROS: Reactive Oxygen Species; Sirt1: Sirtuin 1

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